“It is possible that Omicron Covid-19 also came from the lab’

Some scientists investigating the virus have now concluded that “widespread” genetic mutations are likely to cause Omicron to spread so rapidly in a laboratory setting.

Omicron was first discovered in Botswana, South Africa, and said it may have been brought by a foreign delegation from a country that officials haven’t identified, making it difficult for outside observers to determine the source.

The genetic differences that made Omicron transferable immediately made it seem unlikely to many scientists that it was the result of natural evolution. Some note the “rapid accumulation of mutations in the omicron SARS-CoV-2 variant that made the outbreak possible.”

“There have been a large number of mutations in this variant – much more than we would expect from the natural evolution of this virus,” he said. Virologist Andrew Pekos, professor of molecular microbiology and immunology at Johns Hopkins University’s Bloomberg School of Public Health. “Importantly, many of the mutations occurred in a structural protein — the protein that the virus uses to attach to and enter cells — that is the goal of the vaccine.”

“It did not follow the path of the outbreak,” said a researcher studying the virus for the US government who did not want to be identified because he was not authorized to speak publicly on the subject. “One hypothesis is that there is a cryptic hack, or a completely separate hack,” or a rat-independent “crypto transfer.” Perhaps an infected person will develop weakened immunity, and the active reproduction of the virus takes a very long time.

But there’s another troubling theory—one that some scientists working on the problem see as more likely: that Omicron developed in a lab and escaped.

It’s possible that “the sequencing was done in mice in a lab somewhere, adding”[ed] One scientist familiar with the case says, “The short version is that the least likely scenario is that this was a natural, man-made chain of transmission that we were unable to detect. The number and structure of the changes would require a large number of infections to support it.”

but how?

With Covid so widespread, the statistical odds of lab release are much higher than they were in 2019. In the words of one scientist, “Every lab in the world is now running on Covid. It is highly contagious and risky, even with the best controls. Chance of accidental release. very high “.

“It’s very hard to pinpoint where these things are happening,” he says, “but this is an unprecedented time we’re living in, with so many lab facilities running on the virus at the same time. And so at any time, we’ve run security checks to try and keep pathogens in the lab. But with Having a lot of people working under different conditions, different restrictions, different rules, the potential for a lab to release some work related to COVID is reasonable. It’s something we need to think about.”

If Omicron comes out of the lab, there is a possibility of a horrific or accidental release. Some say additional lab versions of genetically modified versions of Covid are not only possible, but likely.

“suspicious features”

One scientist told me that the traits that seem to distinguish Omicron from the natural evolution of a virus are: “the acquisition of genetic traits that did not exist or have no historical precedent, which appear suddenly in a very short period of time” and “a change in the amount of change or acquisition in a virus beyond what is is expected “.

Scientists have identified at least 45 “acquired omicron mutations” since they diverged from the B.1.1 strain. The mutations represent what some consider to be an unreasonably large number of changes in natural evolution that increased the transmissibility of Omicron.

Another suspicious feature of the omicron, according to many scientists, is the apparent mouse origin.

The truth is that Chinese and American scientists who had been involved in research for years before the pandemic found it very difficult to get the bat coronavirus they were studying to infect lab mice. So the researchers essentially changed lab mice to give them human cells and prescriptions to get them to pick up a genetically modified version of Covid so they could try making vaccines and treatments for it.

The original virus struggled to identify infection in mice and for laboratory use we had to use ACE2 transgenic mice. [also known as] “Humanized mice expressing the ACE2 receptor,” says one of the scientists.

In other words, experts say rodents are not known to naturally pick up and transmit Covid-19, so if Omicron came from a mouse host, it likely happened in an experimental lab.

The Chinese are reporting the same thing. And while US scientists are cautious about the research of Communist Chinese scientists, the fact that they have reached a similar conclusion supports the case.

Evidence for the Worthy scientific paper suggests that (Omicron’s ancestor) jumped from humans to mice, where the conductive mutations “accumulated rapidly,” say authors Changshuo Wei, Ke-Jia Shan, Weiguang Wang, Shuya Zhang and Qing Huan. [quick infection]Then it jumped back to the human.

“We found that the Omicron spike protein sequence showed stronger positive selection than any reported SARS-CoV-2 variants that are known to evolve continuously in human hosts,” the analysis said. The ‘number and frequency of mutations’ was significantly different from the spectrum of viruses that evolved in human patients. †

molecular spectrum (me†e†, the relative frequency of the twelve types of base substitutions) of mutations obtained by the Omicron ancestor was significantly different from the spectrum of viruses that evolve in human patients, but was very consistent with the spectra associated with evolution in the mouse cellular environment. †

From the study:

• The molecular spectrum of omicron mutations prior to the outbreak does not match the evolutionary history of humans
• The molecular spectrum of Omicron mutations before the disease outbreak corresponds to the evolutionary history in mice
• The human SARS-CoV-2 variants reported to date cannot provide the backbone for Omicron.

Experts say human Covid lab experiments on mice may have produced the most incredibly infectious oomicron and somehow reached the general population.

“Natural origin is the least likely scenario,” says one of the leading scientists studying Omicron.

Second lab edition?

If Omicron comes out of the lab, maybe the second version will be just like that.

As I mentioned before, many scientists around the world who have studied Covid-19 from the start said they found traits of human manipulation, strongly suggesting a laboratory origin.

With no cooperation from the Chinese in post-pandemic investigations, and no public accountability or investigation with American researchers, including Dr. Anthony Fauci, who has funded with taxpayer money or conducted research with Communist Chinese scientists, it is not yet clear whether Covid-19 was cooked up in a research laboratory in Wuhan, China. The Chinese never granted access to the lab and documentation needed to establish the truth in the United States or other inspectors, although the United States helped support, inspect and support the lab, and cooperated with laboratory scientists around the world. Risky and dangerous profits. From looking for work with tax money.

In the United States, experts from the US Army Medical Research Institute of Infectious Diseases (USAMRIID) in Ft. Dietrick, Maryland.

Major Jeffrey Kugelman heads the molecular biology department there. As for whether they’ve discovered anything strange so far, Kugelman told me, “There is an ongoing debate about Omicron and we don’t have enough evidence to say, but it hasn’t followed the course of the outbreak.”

“We’re still looking at why the Omicron is different, but it’s very different from what we expected,” Kugelman says.

Additional information about omicron “mass mutations” that cause it to spread more rapidly in humans:

https://pubmed.ncbi.nlm.nih.gov/34968782/

https://onlinelibrary.wiley.com/doi/10.1002/jmv.27516

https://pubmed.ncbi.nlm.nih.gov/35055993/

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